As the crisis over the water in Flint, Michigan, rolls on, we’re learning more and more about the irresponsibility and callousness of officials and politicians in charge.
The mix of austerity politics, environmental racism and sheer ineptitude makes for a shocking brew, yet the physical conditions that have made it literally toxic for Flint residents are neither as exceptional nor as recent as much of the media coverage suggests.
Long before that fateful decision two years ago to turn to the Flint River for the city’s drinking water, pipes made of lead had threaded throughout the city’s underbelly. Flint shares this historical legacy with thousands of other cities, suburbs and towns across our country, and most likely this is not the first time, even in Flint, that these pipes have conveyed tiny amounts of the toxin into homes and children.
Over the past few decades, our environmental laws and agencies have met with much success in curbing some of Americans’ exposure to lead, a damaging neurotoxin. Yet they have struggled to contain this continuing danger precisely because it is literally built into our water systems.
Given that lead has been known as a poison for centuries, why did our forebears in the 19th and early 20th centuries rely on it to carry so vital a fare as drinking water? The answer to this question explains why there are many more Flints waiting to happen.
In the 19th and early 20th centuries, from an engineering standpoint, lead seemed superior to concrete or iron, the alternatives at the time when many municipal water systems were being built. Lead is more malleable and thus easier to bend around corners. It also lasts longer.
Doctors offered virtually no resistance to this decision. After all, they themselves were turning to lead to treat diarrhea or trigger abortions. They recognized only those symptoms of lead poisoning that by today’s standards seem extreme: the severe stomach aches, muscle weakness, kidney failure, seizures and even death that can ensue when lead in the blood rises past 60 micrograms per deciliter – 12 times the current standard.
While lead pipes did occasionally produce “epidemics” this dramatic, health officials remained far more worried about diseases like typhoid, which they knew piped-in water could prevent. As a result, as much as half of the water pipes laid in America’s burgeoning metropolitan areas during the early 20th century were made of lead.
It is also worth noting that lead pipe made up a relatively minor portion of the burgeoning flow of this toxic metal into early 20th-century factories, homes (through paint pigments) and automobiles (through leaded gasoline).
Spurring it along, the lead industry grew rich and powerful. In the time before the advent of the Centers for Disease Control and Prevention (CDC) or the Environmental Protection Agency (EPA), it sponsored its own health research. Some investigators even advanced a thesis that levels of lead in the blood and environment that, in retrospect, seem quite high, were “normal,” a not-so-worrisome condition of modern life.
In fact, the health and behavioral effects of lead from the early to the mid-20th century, as suggested by recent extrapolations from our current knowledge, were likely enormous. It’s estimated leaded pipe alone increased infant mortality by as much as 30 percent in some cities, and led to as much as a 25 percent rise in homicides.
That we have come to know so much more about what lead can do is thus an important part of the story unfolding in Flint.
As investigators of lead’s effects gained greater funding and independence and honed their methods, our understanding of its subtler and longer-term effects grew.
Research on children has shown behavioral disorders, learning difficulties and lowered IQ’s turning up at blood and environmental levels far below what was earlier thought safe. Over the past 30 years, the CDC’s recommended blood levels for lead in the young have dropped precipitously, with no level now acknowledged as really safe.
With greater knowledge of lead’s damaging effects, a concerted campaign against lead started in the 1970s. A ban on its usage in paint in 1978 and a phase-out from gasoline into the 1980s have had considerable impacts.
A 1974 law to control lead in drinking water had less success, however, because it focused on what got pumped into pipes rather than what showed up in people’s faucets.
After an EPA study in 1986 showed one in five of the nation’s drinking water systems carried more lead than considered safe, Congress passed a new Clean Water Drinking Act the same year. This law is still the basis for our current efforts to control the lead that can leach from our water pipes.
Michigan Republican politicians, including Governor Rick Synder, have borne much blame for the Flint crisis – and some of them continue to invite more. But their party was instrumental in the genesis of this act.
It was Ronald Reagan who signed the bill that finally banned the use of leaded pipe and high-lead soldering. And it was George H. W. Bush’s EPA that implemented it, through a 1991 Lead and Copper Rule that required “high-risk residences” to be monitored, with further measures if 10 percent of households exceeded unsafe lead levels of 15 parts per billion (ppb) in their tap water.
Dropping anti-leaching agents
The Clean Water Drinking Act, along with environmental and health officials, did encourage gradual replacement of lead pipes with nontoxic materials such as PVC. But municipalities mainly turned to a chemical fix to lower lead levels, namely anti-leaching agents. Cheaper and faster-acting, these substances could largely prevent lead from entering the water from pipes, soldering and when the source of drinking water changed.
The lead poisoning in Flint recalls a similar water emergency from the early 2000s in Washington, D.C. that highlights the risks of relying on anti-leaching chemicals.
That crisis began in 2001 when the District of Columbia Water and Sewer Authority (WASA) rather suddenly discovered lead levels in its testing that exceeded EPA’s action level.
Events moved even more slowly than in Flint, hitting the headlines only in 2004. Yet the dynamic was similar: those in charge sought to downplay or even suppress what the water testing showed.
The fact was, however, that by 2003 the dimensions of the crisis had become unmistakable. Nearly two-thirds of the water sampled (in “high-risk” homes) exceeded the action level – this in a water system of a half million customers, far bigger than Flint’s.
As with Flint, reports from some homes ranged much higher, upwards of thousands of parts of lead per billion, surpassing levels in wastes deemed officially “hazardous.”
In Washington, D.C., as in Flint, excess lead in faucets owed much to a decision to abandon anti-leaching agents, in this case by the Army Corps of Engineers, whose aqueduct furnished the water for WASA. Cost was part of their rationale, but apparently less so than in Flint; they and the EPA officials who vetted their decision were more worried about high levels of bacteria. What then drew out the lead from existing pipes was a new set of disinfectants also applied by the Army Corps, called chloramines, which had a powerful leaching effect on the lead in the system’s old pipes and joints.
There’s been one big difference between D.C.’s leaded water crisis and that of Flint: the speed and certainty with which the effects have been documented in the blood of water drinkers.
In Washington, an early CDC study failed to find any link between leaded water and blood leads. It was only after the crisis was over that a congressional investigation found the agency to have withheld some critical results. A further study connected D.C.’s water crisis to higher rates of miscarriages and fetal deaths.
In Flint, by contrast, a peer-reviewed study published just last year in the American Journal of Public Health has demonstrated a clear and unequivocal connection between lead levels in the water and those in people’s blood.
What both these experiences make clear is just how risky it has become to rely on monitoring that remains spotty and on chemical treatments, which can be easily abandoned.
We’d now do well to consider the ultimate cause of this type of lead poisoning: the built-in legacy of America’s last leaded century, those old, ever-dangerous conduits by which so many of us still get our drinking water.
Currently, their replacement happens only sporadically, in the wake of crises, if then.
From 2003, the Washington, D.C. government has spent millions digging out and replacing its toxic piping. The mayor of Flint has called for a similar project there, yet so far, promises of support have failed to materialize.
An estimated three to six million miles of lead pipes across our country still carry water, and most all of them are vulnerable to similar dangers, whether at the hands of short-sighted and prejudicial bureaucrats or politicians whose ideology or opportunism leads them to blithely dismiss well-established science.
The best solution would be to replace our lead lines systematically and proactively, not just one crisis-beset city at a time. Until we do so, it’s a safe bet that more Flints lie on our horizon.
Chris Sellers, Professor of History, Stony Brook University (The State University of New York)